Imbalances in growth hormone during puberty can lead to gigantism (growth hormone excesss) or drawfism (growth hormone deficiency) in adults, it can result in acromegaly (growth hormone excess), leading to organs, bones, cartilage, and other tissues to increase in size. Endorphins affect pain perception and euphoria, while growth hormone supports growth by altering metabolism and stimulating growth in bones and muscles. The direct hormone prolactin is usually inhibited from production by dopamine, preventing constant lactation. Thyroid-releasing hormone ( TRH) signals the anterior pituitary to make and release TSH, which stimulates the thyroid gland to make thyroid hormones. Corticotropin-releasing factor ( CRF) also signals the anterior pituitary to make and release ACTH, which in turn stimulates the adrenal cortex to synthesize cortisol. Gonadotropin-releasing hormone ( GnRH) from the hypothalamus signals the anterior pituitary to synthesize FSH and LH, hormones targeting the gonads for gamete production and testosterone synthesis. The PEG hormones, on the other hand, are direct hormones that target non-endocrine tissues, including prolactin, endorphins, and growth hormone. These hormones stimulate endocrine glands to signal the release of another hormone. The FLAT hormones are tropic hormones, including FSH, LH, ACTH, and TSH. The anterior pituitary, located in the front of the pituitary gland, produces and releases seven hormones: FLAT PEG.
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